Those of us who live in Florida’s Collier County (Naples, FL) hear the sound of very low flying planes, usually in the middle of the night. They are flying so low that they make our house shake. When this middle of the night spraying first started, I thought the police were looking for an escapee. But the planes kept coming, middle of the night after middle of the night.
Last night, I finished grocery shopping at about 9PM when I heard the planes. There were people in the parking lot of the supermarket, coming and going. A low flying plane sprayed right over us. I am sickened to spread the word that those living in Naples, FL are being involuntarily subjected to daily/nightly aerial sprays of the neurotoxin: NALED.
The following information comes from E X T O X N E T; Extension Toxicology Network a project of Cornell University, Michigan State University, Oregon Stat University, and University of California (Davis). The support and funding was provided by the USDA National Agricultural Pesticide Impact Assessment program.
Pesticide Information Profile NALED
TRADE OR OTHER NAMES
Trade names include Bromex, Dibrom, Fly Killer-D, Lucanal, RE 4355.
Products containing naled must bear the signal word “Danger” (3).
Naled is moderately to highly toxic by ingestion, inhalation and dermal adsorption. Vapors or fumes of naled are corrosive to the mucous membranes lining the mouth, throat and lungs, and inhalation may cause severe irritation (9). A sensation of tightness in the chest and coughing are commonly experienced after inhalation (14). As with all organophosphates, naled is readily absorbed through the skin. Skin which has come in contact with this material should be washed immediately with soap and water and all contaminated clothing should be removed. Persons with respiratory ailments, recent exposure to cholinesterase inhibitors, impaired cholinesterase production, or with liver malfunction may be at increased risk from exposure to naled. High environmental temperatures or exposure of naled to visible or UV light may enhance its toxicity (9).
The organophosphate insecticides are cholinesterase inhibitors. They are highly toxic by all routes of exposure. When inhaled, the first effects are usually respiratory and may include bloody or runny nose, coughing, chest discomfort, difficult or short breath, and wheezing due to constriction or excess fluid in the bronchial tubes. Skin contact with organophosphates may cause localized sweating and involuntary muscle contractions. Eye contact will cause pain, bleeding, tears, pupil constriction, and blurred vision. Following exposure by any route, other systemic effects may begin within a few minutes or be delayed for up to 12 hours. These may include pallor, nausea, vomiting, diarrhea, abdominal cramps, headache, dizziness, eye pain, blurred vision, constriction or dilation of the eye pupils, tears, salivation, sweating, and confusion. Severe poisoning will affect the central nervous system, producing incoordination, slurred speech, loss of reflexes, weakness, fatigue, involuntary muscle contractions, twitching, tremors of the tongue or eyelids, and eventually paralysis of the body extremities and the respiratory muscles. In severe cases there may also be involuntary defecation or urination, psychosis, irregular heart beats, unconsciousness, convulsions and coma. Death may be caused by respiratory failure or cardiac arrest (9).
Some organophosphates may cause delayed symptoms beginning 1 to 4 weeks after an acute exposure which may or may not have produced more immediate symptoms. In such cases, numbness, tingling, weakness and cramping may appear in the lower limbs and progress to incoordination and paralysis. Improvement may occur over months or years, but some residual impairment may remain in some cases (9).
Naled may cause dermatitis (skin rashes) and skin sensitization (allergies) (2, 6). It is corrosive to the skin and eyes and may cause permanent damage (3). An aerial applicator developed contact dermatitis after using Dibrom. The exposed area became red and felt burned. Later, water filled blisters formed. They became itchy and dry, then flaked off (ACGIH TLVS 4th Ed. & Supplement. 1980).
Repeated or prolonged exposure to organophosphates may result in the same effects as acute exposure including the delayed symptoms. Other effects reported in workers repeatedly exposed include impaired memory and concentration, disorientation, severe depressions, irritability, confusion, headache, speech difficulties, delayed reaction times, nightmares, sleepwalking and drowsiness or insomnia. An influenza-like condition with headache, nausea, weakness, loss of appetite, and malaise has also been reported (9).
Once in the bloodstream, naled may cross the placenta (9).
No information found.
Naled primarily affects the nervous system through cholinesterase inhibition, by which there is a deactivation of cholinesterase, an enzyme required for proper nerve functioning. Lab studies have shown liver damage in rats.
Fate in Humans and Animals
Naled is readily absorbed into the bloodstream through all normal routes of exposure: skin, lungs and gut. Metabolism is in the liver. Accumulation may occur in the bones (of rats). No accumulation effects have been reported in man. Excretion is through the urine (Menzie. Metab. Pesticides. 1969).
Effects on Birds
Naled is highly to moderately toxic to birds. The LD50 for naled in ducks is 52 mg/kg (NIOSH RTECS Online File # 84/8309), 65 mg/kg in grouse and 37 mg/kg in Canadian geese (4).
Effects on Aquatic Organisms
Naled is toxic to most types of aquatic life (8). Some species are especially sensitive to naled (fathead minnow, bluegill, and mosquito fish) (Hndbk Acute Tox. Chem. Fish and Aquatic Inverts. 1980). Agricultural application of 560 g/HA of naled did not kill mosquito fish or tadpoles in irrigation ditches. The 24-hour LC50 for naled in goldfish is 2 to 4 mg/l (3).
Effects on Other Animals (Nontarget species)
Naled is highly toxic to bees (3). Mule deer are more resistant than most wildlife species. The LD50 for naled in mule deer is 200 mg/kg (4).
Breakdown of Chemical in Water
Naled is rapidly broken down in water. The half-life is about 2 days (even though naled is practically insoluble in water). Naled is moderately volatile.
Naled may produce a pollution hazard if dilution water is improperly disposed of, or if run-off from fire control is not properly contained (11).
Breakdown of Chemical in Vegetation
The organophosphate class of insecticides tends to be readily taken up and metabolized by plants. They tend to have short half-lives in the soil and do not carry over through successive plantings.
Plants breakdown naled and DDVP is created by the debromination of the naled molecule in a 1:1 relationship. These can be further metabolized or evaporated off (17).
Check out what Puerto Rico’s governor did when he found that Naled had been shipped from the US to Puerto Rico to combat the Zika virus.
Realize that no one has protected us from being sprayed up to 2 times/day.
I’ll be leaving Naples, FL.
Between the daily toxic spraying and the polluted Gulf water caused by regular releases of toxic water from Lake Okachobee that finds its path to the Gulf killing marine vegetation and sea life, turning the once crystal blue water to murky, brown colored, smelly, possibly contaminated beach water….there is no good reason to stay here.
I just want to make a public statement about this. My next door neighbor was not even aware of the Naled spraying and a woman who sat next to me on a plane exclaimed “What happened to the water?” when we flew over the brown, murky water that is now the truth in “Beautiful Naples.”
So sad to see what is being done to our planet, our home, our lives…….